Could sustained immune pressure on viral transmissibility eventually lead to the natural selection of virulence-enhancing changes in viral glycosylation?

I have previously expressed my amazement at the remarkable resilience of complex biological systems, such as the mammalian immune system, in mitigating and/or postponing the severe consequences of C-19 vaccine-induced viral immune escape on human health (https://www.voiceforscienceandsolidarity.org/scientific-blog/to-whom-it-may-concern). This resilience seems particularly applicable to viral immune escape mechanisms that threaten the survival of the host species. I have proposed that mutations in the glycosylation pattern of SARS-CoV-2 could eventually drive viral evolution toward enhanced virulence, potentially resulting in rapid death (https://www.voiceforscienceandsolidarity.org/scientific-blog/predictions-gvb-on-evolution-c-19-pandemic). It is, therefore, reasonable to investigate whether evolutionary changes in the virus’s glycosylation profile could also contribute to attenuating viral virulence and/or delaying an explosion in mortality rates in highly C-19 vaccinated populations.